Serum amyloid A and mitochondrial DNA in extracellular vesicles are novel markers for detecting traumatic brain injury in a mouse model

Tony Z. Tang, Yingxin Zhao, Deepesh Agarwal, Aabila Tharzeen, Igor Patrikeev, Yuanyi Zhang, Jana DeJesus, Stefan H. Bossmann, Balasubramaniam Natarajan, Massoud Motamedi, Bartosz Szczesny

Research output: Contribution to journalArticlepeer-review

Abstract

This study investigates the potential use of circulating extracellular vesicles’ (EVs) DNA and protein content as biomarkers for traumatic brain injury (TBI) in a mouse model. Despite an overall decrease in EVs count during the acute phase, there was an increased presence of exosomes (CD63+ EVs) during acute and an increase in microvesicles derived from microglia/macrophages (CD11b+ EVs) and astrocytes (ACSA-2+ EVs) in post-acute TBI phases, respectively. Notably, mtDNA exhibited an immediate elevation post-injury. Neuronal (NFL) and microglial (Iba1) markers increased in the acute, while the astrocyte marker (GFAP) increased in post-acute TBI phases. Novel protein biomarkers (SAA, Hp, VWF, CFD, CBG) specific to different TBI phases were also identified. Biostatistical modeling and machine learning identified mtDNA and SAA as decisive markers for TBI detection. These findings emphasize the importance of profiling EVs’ content and their dynamic release as an innovative diagnostic approach for TBI in liquid biopsies.

Original languageEnglish (US)
Article number108932
JournaliScience
Volume27
Issue number2
DOIs
StatePublished - Feb 16 2024

Keywords

  • Cellular neuroscience
  • Molecular neuroscience

ASJC Scopus subject areas

  • General

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