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Aditi, PhD
Asst Prof (N-T Track Research)
,
Microbiology And Immunology
Email
fnaditi
utmb
edu
2013
2024
Research activity per year
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Research output
(13)
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(6)
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Dive into the research topics where Aditi is active. These topic labels come from the works of this person. Together they form a unique fingerprint.
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INIS
humans
100%
messenger-rna
99%
proteins
88%
exports
87%
diseases
82%
mice
49%
coordinates
47%
stimulation
46%
dna damages
45%
yeasts
44%
human cells
44%
control
44%
brain
42%
mutations
40%
genes
39%
instability
35%
rna
33%
defects
31%
dna
31%
interferon
31%
inflammation
31%
dynamics
30%
interactions
29%
pools
27%
cytokines
26%
nervous system
26%
kinases
24%
typhus
22%
promoters
22%
glycogen
22%
architecture
22%
phosphorylation
22%
plasticity
22%
modulation
22%
mitogens
22%
gene loci
22%
chromatin
22%
methylation
22%
excision repair
22%
growth factors
22%
tumor necrosis factor
22%
receptors
22%
malaria
22%
fibroblasts
22%
pathogenesis
20%
losses
19%
particles
18%
production
15%
data
14%
neurons
13%
Biochemistry, Genetics and Molecular Biology
Messenger RNA
80%
Protein
78%
Stress Granule
57%
Human Cell
44%
Yeast
44%
Isoform
27%
DEAD Box
27%
Dynamics
24%
Mitogen-Activated Protein Kinase
22%
Genome Instability
22%
Interferon Type I
22%
Drive
22%
Nucleotide Excision Repair
22%
Modulation
22%
Glycogen Synthase Kinase
22%
Phosphorylation
22%
Ribonucleotide
22%
RNA
17%
C-Terminus
17%
Ribonuclease
16%
ATPase Assay
16%
Gene
15%
Coiled Coil
14%
Association
12%
Nuclear Pore Complex
11%
DNA Damage
11%
N-Terminus
11%
Stress
11%
DNA
11%
ATPase
9%
Regulatory Mechanism
8%
Inositol
7%
Cell Stress
7%
Glycogen Synthase Kinase 3
7%
Kinase
7%
Gene Expression
7%
Epigenetic Repression
5%
Plasmid
5%
Peptide
5%
Nucleic Acid Metabolism
5%
Electron Microscopy
5%
Insect Cell
5%
Phenylalanine
5%
Translation Termination
5%
Proline
5%
Fluorescence
5%
Interferon
5%
Glutamine
5%
Non-Homologous End Joining
5%
Oligomerization
5%
Keyphrases
Dbp5
22%
Inflammation
22%
Gle1
22%
Stress Granule Dynamics
22%
Genome Integrity
22%
IP 6
22%
Sickness Behavior
22%
Nervous System
22%
Ribonuclease H2
16%
Post-acute Sequelae of COVID-19 (PASC)
16%
Innate Immune Signaling
14%
Neuronal Cells
14%
T-box Expressed in T Cells
14%
Neurological Diseases
14%
DNA Damage
14%
Neuroinflammation
14%
Chromatin Conformation
11%
Aicardi
11%
Future Therapies
7%
Neuropathology
7%
Immune Activation
7%
Genome Maintenance
7%
Maintenance Mechanism
7%
DNA Repair Deficiency
7%
Human Disease
7%
System Function
7%
Neurodegeneration
7%
Nervous System Development
7%
ITPR1
5%
GRID2
5%
RNASEH2B
5%
Type I Interferonopathy
5%
RNase H2
5%
Inflammation-related Pathways
5%
Transcriptome Dynamics
5%
Defense Response
5%
Impaired Cognition
5%
Viral Variants
5%
Longitudinal Behaviour
5%
SARS-CoV-2 Delta Variant
5%
